Cayce
Comprehensive
Symptom
Inventory
(CCSI)
Workbook and Manual
Version
1.0
SCALE 13
STOMACH POSITION
CONTENTS
EXPLANATION
In over 120 readings, Edgar Cayce cited abnormal
stomach position as a contributing factor to serious systemic imbalances.
Typically, the readings use expressions such as "tilted," "tipped," or
"dropped" when referring to abnormal stomach position. In general,
two patterns of abnormality are discussed:
-
The stomach position may allow food to pass through too quickly without
allowing proper digestion. The stomach is too vertical with the upper
portion (cardiac) too high or the lower end (pyloric too low).
-
The stomach position may hold foods too long producing too much fermentation.
The upper end (cardiac) is dropped or the lower end (pyloric) is raised.
Abnormal stomach position causes or contributes to:
-
Poor digestion and assimilation,
-
Poor eliminations as poorly digested food is passed into the intestinal
system,
-
Disturbed circulation,
-
Abnormal acid/alkaline balance,
-
Abdominal symptoms including stomach pain, feelings of heaviness after
eating, etc.
-
Unbalancing of hepatic system functioning, primarily the liver, resulting
in systemic toxicity and related problems.
Abnormal stomach position (and misplaced abdominal organs
generally speaking) were well known problems among osteopathic physicians
and medical doctors during the late nineteenth and early twentieth centuries.
The latter portion of this section contains selections from this early
literature, including:
THERAPEUTIC OPTIONS
Here are some therapeutic interventions frequently
recommended by Edgar Cayce in cases involving abnormal stomach position.
MANUAL THERAPY
One of the most frequent therapeutic recommendations
for abnormal stomach position was spinal manipulation and massage.
Special attention should be given to the nerve centers which provide impulse
to the stomach at the second to six thoracic vertebrae. Direct abdominal
manipulation to reposition the stomach may be helpful if the clinician
has been trained in such techniques.
STOMACH SUPPORT
A support belt or bandage was often recommended to
help reposition the stomach. For example, in reading 3607-1, a woman
was told to "use an elastic belt to support the abdomen and the stomach
itself. Or still more preferable, though it would be much more trouble
to use, bind heavy gauze about the body. This would necessarily be
done daily, and that it be taken off when the treatments of the osteopath
are given." For severe cases, the belt or support may need
to be worn for several weeks or until a stomach position has been achieved.
EXERCISE
In addition to moderate general exercise (such as
walking), specific exercises were sometimes suggested to strengthen the
muscles supporting the stomach. Reading 848-1 provides typical recommendations
for exercise and support belt to correct abnormal stomach position.
This reading suggests each morning and evening the individual bend forward
with a circular motion of the body, circling the body for 5 - 15 minutes.
ELECTRIC VIBRATOR
The electric vibrator was recommended in several
readings for persons with "dropped stomach." In these cases,
food often stays in the stomach too long producing an uncomfortable sensation.
Using an electric vibrator directly over the stomach can stimulate peristaltic
action and assist the stomach in moving the food along. Also, use
of the electric vibrator along the spine (especially at the 4th, 5th, and
6th thoracic centers) may be helpful in stimulating the nerve centers governing
the stomach.
REST
To balance the exercise and assist with healing,
periods of rest while in a reclining position with the feet raised above
the stomach was sometimes suggested.
DIET
Usually a balanced diet with an emphasis on alkaline-producing
foods. If the individual is suffering from general debilitation and
low vitality due to poor assimilations, vegetable and beef juices may be
helpful.
DIGESTIVE AIDS AND NUTRITIONAL SUPPLEMENTS
A variety of digestive aids and nutritional supplements
were recommended to address the functional imbalances related to abnormal
stomach position. For hypoacidity, Acigest (a product containing
hydrochloric acid) was sometimes prescribed (usually taken in conjunction
with Calcios, a calcium supplement). Other frequently recommended
digestive aids included lactated pepsin and milk of bismuth, and Alcaroid.
Yellow saffron tea and slippery elm bark water were prescribed in several
cases involving abnormal stomach position.
HYDROTHERAPY
Frequently, digestive problems originating in the
stomach compromise the functioning of the entire intestinal tract resulting
in constipation and other chronic bowel problems. Colonic irrigation
was recommended in numerous readings for persons with abnormal stomach
position to assist in cleansing toxins from the lower GI tract.
FURTHER ASSESSMENT
The abnormal stomach position scale may be used for
screening for further assessment. Further assessment options may
include:
-
Abnormal stomach position may be assessed by careful palpation of the abdomen.
-
Imaging techniques (such as barium swallow and ultrasound) may be helpful
in assessing for this problem.
-
History of stomach injury (such as a blow to the stomach) or spinal injury
to the second to sixth thoracic vertebrae may be significant.
-
Physical examination of the spine with special attention to the second
to sixth thoracic vertebrae may also be useful in locating somatic dysfunction
associated with abnormal stomach position.
DOCUMENTATION
SCALE 13: ABNORMAL STOMACH POSITION
SYMPTOM
|
READINGS
|
Abnormal appetite (increased, decreased, or erratic) |
5599-1, 3607-1, 2374-1, 2243-1, 1149-1, 1048-2, 1001-1,
888-1, 848-1, 667-8, 565-1, 562-1 |
Indigestion or high acidity in stomach, throat, or mouth |
5641-1, 5261-1, 4722-1, 4456-1, 4276-1, 3705-1, 2370-1,
2356-1, 1259-1, 1143-1, 931-2, 829-1, 730-1, 728-1, 667-1, 565-1, 263-1 |
Stomach or intestinal gas |
5210-1, 4631-2, 3705-1, 2395-1, 2290-2, 1419-2, 1216-1,
810-1, 694-1, 667-8, 313-6, 268-1, 130-1, 39-1 |
Abnormal heart action (low or high) or discomfort around
heart |
5590-1, 4722-1, 4631-2, 4456-1, 3742-1, 2370-1, 2360-1,
2352-1, 1048-1, 848-1, 667-1, 565-1, 357-1, 267-2, 263-1, 130-1, 39-1,
6-1 |
Stomach empties too slowly or too quickly after eating |
5210-1, 5116-1, 4372-1, 4276-1, 2072-14, 1161-1, 1028-1,
943-1, 667-8, 544-1, 263-3 |
Headaches |
5641-1, 5599-1, 5261-1, 5210-1, 4403-1, 2374-1, 2243-1,
1882-1, 1387-5, 943-1, 848-1, 811-1, 614-1, 562-1, 482-3, 451-1 |
Nausea |
5599-1, 5210-1, 4631-2, 4403-1, 4318-1, 3742-1, 1065-1,
1001-1, 848-1, 811-1, 562-1, 482-3, 377-1, 357-1, 274-5, 263-11 |
Constipation |
4631-2, 4318-1, 1259-1, 1048-2, 888-1, 843-1, 728-1,
544-1, 268-1 |
TRADITIONAL OSTEOPATHIC PERSPECTIVE
[NOTE:
The following discussion of "enteroptosis" which includes dropped stomach
(gastroptosia) comes from The Practice and Applied Therapeutics of Osteopathy
by Charles Hazzard, D. O. which was published in 1905.]
ENTEROPTOSIS
Enteroptosia is a disease in which various of the
abdominal and pelvic viscera leave their natural positions, slipping downward
into the abdominal and pelvic cavities. It is a common and distressing
complaint, frequently overlooked or not recognized. It is sometimes
regarded as a symptom group, but may, from the osteopathic point of view,
be regarded as an idiopathic condition, due to specific lesion.
These cases are often treated for some one feature,
as for nervous dyspepsia, constipation, operation for floating kidney,
etc. It is a common error to overlook the essential condition of
the disease. The Osteopath who gives close attention to a class of
neurasthenic, flat-chested, constipated patients, who complain of lack
of bodily and mental vigor, many and various indefinite nervous symptoms,
abdominal pulsation, vaso-motor disturbance, etc., will find most interesting
material. The multitude of symptoms may vary greatly in different
cases, but the presence of neurasthenic conditions, altered thorax and
spine, and unnatural abdominal condition, either of walls, viscera, or
both, will usually afford an unmistakable sign of the disease. After
a little experience with such cases one learns to recognize them at a glance
when presented for examination. Once seen these cases can hardly
be mistaken, and a few moments examination reveals a story of disease beginning
imperceptibly, the growing conviction through many months or some years
that something was wrong, the attempt to seem well because no decided disease
seemed present, or a long course of treatment for various ills, none of
which reached the true condition. This most common disease it still
but seldom clearly recognized or intelligently handled.
LESIONS AND CAUSES: The common description of its
etiology is unsatisfactory. Tight lacing, traumatism muscular strain,
and repeated pregnancies are mentioned. The condition of relaxed
abdominal walls and prominent viscera due to repeated pregnancies may probably
be rightly regarded as a separate condition. It is due to a physiological
act, and does not present those specific lesions nor the resulting symptoms
found in neurasthenic enteroptosis. Tight lacing, traumatism, and
muscular strain may produce those lesions found to be the cause of such
conditions.
These cases commonly present spinal, rib, diaphragmatic
and abdominal lesions. Spinal lesions may be of any of the kinds
found in the spine ordinarily, and may occur anywhere along the splanchnic
or lumbar region. Rib lesions may occur in any or all of the lower
six ribs on either side.
Mobility of the tenth rib is regarded by a German physician, Dr.
B. Stiller, (Phil. Med. Journal, Jan. 13, 1900,) as a pathognomonic
cause of enteroptosis (Boston Osteopath, Jan. 14, 1900). Undoubtedly
it could interfere with the sympathetic connections of the abdominal viscera
and become a factor in causing this condition. But, from an osteopathic
viewpoint, lesions of other ribs, and of spinal vertebrae, etc., may be
as potent in producing the "basal neuropathy" concerned in this disease
as its fundamental pathological condition. Further, rib lesions may
cause a condition of the diaphragm in which its normal tone is lost, and
prolapse in it causes ptosis in the abdominal organs which it aids in supporting.
Spinal lesions may participate in causing the atonic condition of the diaphragm.
Spinal and rib lesions, aside from derangement of
the diaphragm, act to produce enteroptosis by interfering with the spinal
sympathetic connections of the viscera and of their omental supports.
Impeded circulation and nerves supply, vaso-motor, motor, secretary, trophic
and sensory produces at the same time derangement of function in the organs
and weakness in their mesenteric supports. These conditions work
together to bring about the disordered function and the displacement of
these organs. The displacement of itself furthers the present bad
conditions by mechanically interfering with the activities of organs, stretching
nerve fibers and blood vessels which are carried in the now elongated omenta,
kinking the colon at various points, etc. The viscera, having sunk
down into the abdominal cavity, cause prominence of the lower abdomen,
leaving a hollow in the upper abdomen, thus giving to it the peculiar boat-shaped
appearance described as "scaphoid abdomen."
Lower dorsal and lumbar lesion may interfere with
the spinal innervation of the abdominal walls, cause them to lose their
tone and to dilate. Intra-abdominal pressure is thus lessened and
the organs are allowed to prolapse.
According to Byron Robinson, enteroptosis begins
with a weakening of the abdominal sympathetic, which loses its normal power
over circulation, secretion, assimilation and rhythm. That this weakness
of the abdominal sympathetic and its consequent loss of function originates
in spinal lesion to its origin in the splanchnic nerves has already been
pointed out and fully discussed in considering the diseases of the stomach
and intestines, q. v. The anatomical relation of such lesions to parts
affected was pointed out.
The PROGNOSIS in these cases is very favorable,
but the progress of the cure is likely to be slow. Generally improvement
begins immediately upon treatment and may progress to a cure in a few months.
Other cases yield more slowly, though relief is soon given, and require
an extended course of treatment to effect a cure.
The TREATMENT must be both constitutional and local.
The latter consists in the removal of lesion and in abdominal treatment.
Lesions anywhere to the splanchnic and lumbar regions, to the ribs, thorax
and diaphragm, must be treated after their kind, according to directions
given in Part I. With spine, ribs, and diaphragm restored to normal condition,
the underlying causes of the enteroptosis have been removed. Corrected
nerve and blood supply to the organs and their supports aids in correcting
their function and strengthens the supporting tissues to hold them in place
when restored by abdominal manipulations.
Correction of spinal lesion also aids in restoring
nutrition and tone to the relaxed and atrophied abdominal walls.
This process is furthered by a thorough treatment upon the abdominal walls.
This renders the use of the favorite abdominal bandage unnecessary, and
it is gradually laid aside. Throughout the course of the case the
restored abdominal walls act as the bandage has done to hold the organs
to their places as replaced by the treatment. With corrected spine,
free blood and nerve supply to all the visceral supports, and a strengthened
abdominal wall, no difficulty is found in getting the parts to gradually
be retained in their normal positions. Thorough spinal stimulation
over the splanchnic and lumbar; areas is kept up for the purpose of increasing
the blood and nerve supply to the parts in question.
Abdominal work, aside from treatment of the walls,
is directed to raising and replacing the viscera. This is readily
accomplished by various treatments. (II, III, IV, Chap. VIII.) This
releases and renews circulation and nerve supply at the same time, removes
pressure of organs upon each other, gives freedom of motion, and aids in
strengthening the omenta to hold the parts in place.
The diaphragm has been restored to normal position, and tone by correction
of those lesions originally deranging it.
The constitutional treatment must be thorough and
general to restore the patient from the nervous, circulatory, nutritional,
and other effects of the disease. A most thorough general spinal
treatment must be given. Thorough stimulation of heart and lungs,
treatment of the cervical sympathetic, and attention to kidneys, liver
and skin accomplishes the desired object. The auto-intoxication usually
present is overcome by this treatment of the excretory organs. The
constipation, dyspepsia, and other functional disorders are corrected by
the restoration of the organs concerned.
The patient should be much out of doors, free from
worry, and careful not to become fatigued. Deep breathing exercises
are beneficial.
THE ABDOMINAL AND PELVIC BRAIN
[NOTE:
The following discussuion of "enteroptosis" which includes dropped stomach
(gastroptosia) comes from The Abdominal and Pelvic Brain by Byron
Robinson, M. D. which was published in 1907.]
Gastroptosia.
Gastroptosia or atonia gastrica signifies abdominal
relaxation. It includes distalward movement of the stomach and relaxation
of the abdominal wall. It is a part and parcel of splanchnoptosia.
Gastroptosia (or its equivalent atonia gastrica) practically includes the
terms dilation of stomach, ectasis ventriculi, insufficiency of the stomach,
gastric insufficiency, motor insufficiency, ischochymia (retention of cbyme),
myasthenia, extasis gastrica, because it signifies abdominal relaxation
and relaxation includes dilatation and motor insufficiency. Therefore
gastroptosia is a proper, comprehensive, scientific term which signifies
ptossi, dilatation and motor insufficiency of the stomach. Gastroptosia
is of paramount importance to physicians as its existence is frequent in
every day practice.
In early embryonic life the stomach is absolutely
vertical and the child is practically born with a vertical stomach and
besides I have observed scores of permanently vertical stomachs in adult
autopsies (perhaps from arrest of development). With the growth of
the child the stomach rotates following the atrophying liver. In
the adult the rotated stomach is supplied on its ventral surface (left)
by the left vagus and on the dorsal surface (right) by the right vagus.
Food aids by its weight and distention to force the stomach distalward.
In gastroptosia the lesser curvature and pylorus moves distalward.
Fig. 186. This illustration presents the horizontal
stomach, which in gastroptosia dilates from pylorus to cardiac extremity
and passes distalward as in Fig. 184 - a gastro-duodenal dilatation.
Sig. represents the sigmoid flexure in a 180 deg. condition of physiologic
volvulus. |
Etiology.
Gastroptosia arises from a variety of causes.
Disordered respiration with consequent descensus of the diaphragm and distorted
distal thorax (ribs) is among the first disturbances. In short gastroptosia
coexists with splanchnoptosia. Gastroptosia may be due to an abnormally
distalward location of the diaphragm. In hepatoptosia the liver forces
the pylorus distalward and to the left. Relaxed abdominal walls, rapidly
repeated pregnancies, infected puerperium (i. e., practically subinvolution
of the abdominal wall), compression from waist bands, liver or spleen tumors,
pleuritic effusions or adhesions, pericarditis are fruitful causes of gastroptosia.
I have observed in autopsies that peritoneal and especially omental adhesions
play an extensive role in gastroptosia.
Gastroptosia is congenital or acquired. The
acquired gastroptosia is discernible in the change in normal relations
of the space in the proximal abdomen and distal thorax especially in the
manifestation of respiration. In general we observe at post mortems
two forms of gastroptosia, viz.: (a) the whole stomach appears (with the
lesser curvature and pylorus with a transverse position) moving caudal,
(b) the distalward moving stomach assumes more or less a distinctly vertical
position.
I wish to state, that, from personal autopsic observation
in the abdominal viscera in over 700 subjects, the stomach varies extensively:
(a) in position, (b) in dimension, (c) in form.
Gastroptosia may be due to constitutional defects
or anomalies in both sexes. The peculiar formed chest, as funnel
shaped, chicken breast, may be observed in subjects with gastroptosia which
is part and parcel of splanchnoptosia. Gastroptosia occurs in subjects
with tubercular habitus - constitutional defects.
Fig. 187. This represents a vertical
stomach. During gastro-duodenal gastroptosia the chief gastric dilatation
occurs at the distal end of the stomach. The superior mesenteric,
S, compressing the transverse duodenum, causes the gastro-duodenal dilatation.
This figure presents a non-descended cecum, and an ileum, 1, adherent
to the ileopsoas muscle. 1, 2, 4 representing the dorsal insertion line
of the meso-sigmoid. |
Gastroptosia (or splanchnoptosia) does not as a rule
occur in strong robust subjects. Obvious stigmata of degeneracy accompanying
splanchnoptosia subjects with elongated narrow thorax are liable to gastroptosia
because the diaphragm occupies an abnormally distalward location.
Pulmonary emphysemia or pleural effusions force the diaphragm distalward
favoring gastroptosia (and concomitant splanchnoptosia). Mechanical
conditions may enhance stomachoptosia as supraumbilical hernia, inguinal
or femoral hernia, peritoneal adhesions. Rapidly repeated gestations
present a large field of gastroptosia so fully discussed by Landau as well
as rapid loss of large quantities of fat. In multipara and subjects
with loss of quantities of flesh the abdominal muscles become relaxed and
lose their delicate active poise in maintaining the viscera in their normal
physiologic position.
Gastric Dilatation in Splanchnoptosia.
Many times I have observed in autopsy extensively
dilated stomach, the existence of which in life had not been suspected,
first, because the physical condition of the patient was favorable and
second compensatory action between stomach and pylorus was still favorable.
A relation exists between the dimensions of the pylorus and that of the
stomachs compensatory action. I performed an operation on a woman
who had vomited for years with a dilated stomach. In this case the
pylorus had dilated slightly and its flexion increasing by ptosis obstructed
the free evacuations of the stomach contents.
Fig. 188. illustrates gastroptosia.
The colon transversum forced distalward into the pelvis by the stomach.
1, liver with hepatoptosia; 2, stomach in the lesser pelvis; 3, 4, duodenum
dilated; 5, the jejunum, normal caliber; 6, transverse colon. This
cut represents gastro-duodenal dilation-the second stage of splanchnoptosis.
The artist neglected to present the duodenum dilated. |
Again we note autopsies in which a subject possesses
a markedly dilated stomach with slight difficulty in evacuation of stomach
contents through the pylorus because the pyloric ring had dilated proportionately
with the stomach dilatation allowing free evacuation, free passage of food
from stomach to duodenum, free drainage - here is compensatory dilatation
- of stomach and pylorus, resembling that of the cardiac valves, however,
suddenly the stomach and pyloric compensatory action may fail and the patient
passes swiftly onward and swiftly downward - exactly as in valvular heart
lesions.
The etiology of gastroptosia may be sought chiefly
in constitutional defects. However, mechanical derangement is sufficiently
obvious in gastroptosia. Gastro-duodenal dilatation which plays such
an extensive role in splanchnoptosia will be discussed and illustrated
in a future chapter. Combined gastro-duodenal dilatation due to the
compression of the transverse duodenal segment by the superior mesenteric
artery vein and nerve is a frequent condition and though I have published
articles on it for a decade it is still but limitedly recognized.
The symtomatology may be practically negative or of the most aggravated
kind. It may be stated, in general, that gastroptosia is without
symptoms so long as the stomach functionates normally which mainly prevails
while the subject is in favorable physical condition. Gastroptosia
presents symptoms when detention and composition food occurs and general
infection results. Indirect symptoms may arise as in splanchnoptosia,
e. g., fatigue, debility, constipation, insomnia. Meinert insist
that gastroptosia is a common cause of chlorosis.
The symptoms of gastroptosia are generally proportionate to the degree
of stomachic dilatation. Kussmaul originally observed that gastroptosia
is frequently accompanied by a disturbance of the motor nerves of the stomach.
This may be due to the trauma traction on the vagi from change of gastric
position. Gastroptosia frequently coexists with multiple nervous
symptoms, but the nervous symptoms may be due to splanchnoptosia. However,
gastroptosia is a disease and is liable to be accompanied by disturbed
motion, absorption, secretion and sensibility of the stomach. Change
of form and position of the stomach may not lead to any more nervous symptoms
than change of form and position of the uterus, however dislocation of
the uterus, i. e., permanent fixation, is the result of some disease.
Malposition of the stomach does not produce neurasthenia
any more than malposition of the uterus. The position of a mobile
viscus is not responsible for neurosis, for mulitple positions or multiple
deviations must not be considered abnormalities. It is disease that
produces neurosis, not position of viscera. Original disease which
produced the malposition of the viscus should be held responsible for the
nervous disturbance. Again there can be no doubt that the symptoms
of gastroptosia and nephroptosia are constantly mistaken for each other
especially by the careless examining surgeon with a tendency to nephropexy.
There is no characteristic stomach contents peculiar to gastroptosia.
In gastroptosia pain is generally prevalent in the proximal abdomen and
'Lumbar regions. It is true that subjects with gastroptosia (a part
and parcel of splanchnoptosia) present multiple neurotic symptoms simulating
disturbed mobility, secretion, absorption and sensibility of the stomach.
However, this may belong in the congenital debility or predisposition of
the patient - due to the disturbance created by anatomically dislocated
viscera and consequently pathologic physiology. Gastroptosia increases
the weight of the stomach.
Diagnosis.
Gastroptosia is less recognized than nephroptosia
which is diagnosed with more facility and besides the pexyites are more
vigorously in search of nephroptotic victims.
Percussion and auscultation with various quantities
of fluid in the stomach may suggest the position and dimension of the stomach.
Palpable epigastric pulsation, absence of projecting
abdominal wall in the epigastrium and projecting abdominal walls in the
hypogastrium aid in the diagnosing gastroptosia.
The most exact method to determine the position and
dimension of the stomach is by inflation, viz.: (a) by generation of gas
within the stomach. The most frequent method of gastric inflation
practiced is by directing the patient to drink a glass of water containing
some sodium bicarbonate and immediately to drink another glass of water
containing tartaric acid whence carbonic acid gas is formed distending
the stomach by air. (b) Another method to inflate the stomach is by introducing
into the stomach a tube whence air is forced through it for distention,
whence its form, position and dimension may be observed through the abdominal
wall. (c) A third method of diagnosing the form, position and dimension
of the stomach is by distending the stomach by fluid.
When the major curvature is at or below the umbilicus
and the pylorus and lesser curvature have moved distalward the diagnosis
of gastroptosia is confirmed. A healthy stomach maintains the position
of its borders regardless of the subject's attitude. In gastroptosia
the borders of the stomach change according to the patient's position.
In gastroptosia with the patient in the erect posture the major stomach
curvature and pylorus will be more caudal, while if the patient's posture
is recumbent the pylorus and major curvature cephalad. Succussion
(splashing sound) is a method to diagnose gastroptosia by agitating air
and water in the stomach through shaking the body. The splashing
sound may also be obtained by palpating the stomach while the patient is
in the recumbent position. A splashing sound elicited from the stomach
means practically gastroptosia-relaxation, atony. Some persons by
practicing pressure of the abdominal muscles of the stomach can produce
various sounds in the stomach. Such persons perhaps possess abnormally
a large stomach and powerful abdominal muscles, however, like a fakir have
exaggerated an anomaly. Gastroptosia may be diagnosed by transillumination,
i. e., introducing an electric light in the stomach whence its contour
may be observed. This method was advocated in 1845 by Casenave, later
in 1867 Milliot improved it by experimentation, however, Dr. Max Einhorn
of New York practically first made successful use of (the gastrodiaphane)
transillumination of the stomach in man and demonstrated the utility of
gastro-diaphanes copy.
Inspection may present a depression in the epigastrium
and a projection in the umbilical region. This method of diagnosis
may be sufficient in spare persons to announce gastroptosia. The
X-ray may be used to note the position of the stomach by administering
substances which will cast a shadow, as subnitrate of bismuth or metallic
salts administered in capsules. Treatment is medical, mechanical,
surgical.
Treatment.
1. The medical treatment consists in regulation
of diet and function. The dietetic management consists in administering
limited quantities of prescribed food at regular three-hour intervals.
The diet should be cereals, vegetables, milk and eggs. All high seasoned
food, pastry, pie, cake, spices, meat should be excluded to avoid fermentation.
The most essential medical treatment consists in
"visceral drainage" as ample sewerage the evacuating channels should be
flushed. Gastroptotics may live healthy with ample visceral drainage.
The tissues and tissue spaces in gastroptosia (splanchnoptosia) require
flooding, washing, so that the subject may be free from waste laden blood
and residual debris. Every evacuating visceral tract (tractus intestinalis,
perspiratorius, urinarius respiratorious) should perform maximum duty.
The sheet anchor treatment for gastroptosia is regulation of food and fluid,
and maximum sewerage of visceral tracts. Dietetics, hygiene, anatomic
and physiologic rest, properly supervised tend extensively to the welfare
in the life of a splanchnoptotic.
Fig. 189 illustrates the third stage
of splanchnoptosia, viz.: gastro-duodenal dilatation. It shows the transverse-colon
(5) in the lesser pelvis. The widely dilated stomach (1) is drawn leftward
by hooks (10) from its bed to show the duodenum (2) dilated by the superior
mesenteric artery, vein and nerve, (3) 4, the normal calibered loops of
enteron; 6, right colon; 7, cecum; 8, the appendix. Note the enteron
loops crowded into the lesser pelvis. |
Mechanical treatment in gastroptosia judiciously
applied affords wonderful relief. Stomachic irrigation occasionally
renders much comfort. The treatment consists in the application of
abdominal wall to support the viscera. This is accomplished by various
kinds of abdominal binders - elastic and non-elastic. I use sometimes
an abdominal binder within which is placed a pneumatic rubber pad which
is distended with air to suit the patient's comfort. Dr. E. A. Gallant
employs a suitable fitting corset. The adhesive strapping method
of Achilles Rose is practical, rational and economical and affords excellent
relief. The recumbent position aids the patient. The mechanical method
attempts the forcible reposition of the stomach to its physiologic position
and there to maintain it by aids applied to the abdominal wall - a rational
method. Pregnancy practically relieves the gastroptosia for a season.
Splanchnoptotics experience more comfort from rational adhesive strapping
(mechanical supports) than from surgical procedures.
Surgical treatment in gastroptosia is a very limited
field. It espouses two methods, viz.: (a) The surgery is applied
to the stomach itself as gastro-enterostomy, the Heinicke-Mickulicz operation
(both tend to cure by visceral drainage) the replication of the stomach
parietes or the attempt to shelve the stomach by omentum or mesentery (both
unphysiologic, irrational). (b) The abdominal wall is employed
to support the stomach as by incision and over-lapping like a double breasted
coat, or by enclosing, uniting the two musculi recti abdominales in one
sheath. Both methods attempt to relieve by lessening the abdominal
cavity and forcing the stomach into its normal physiologic position (both
rational). A third method is to perform gastropexy, i. e., suture
the stomach to the abdominal wall (limited, irrational in general).
|